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phospho ire1α p ire1α nb100 2323 antibodies  (Novus Biologicals)


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    Novus Biologicals phospho ire1α p ire1α nb100 2323 antibodies
    Phospho Ire1α P Ire1α Nb100 2323 Antibodies, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 96/100, based on 231 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/phospho+ire1%CE%B1/pm41330741-67-0-10?v=Novus+Biologicals
    Average 96 stars, based on 231 article reviews
    phospho ire1α p ire1α nb100 2323 antibodies - by Bioz Stars, 2026-07
    96/100 stars

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    SB supplement ameliorated TMAO-mediated activation of ER stress signaling and dysregulation of ionic signaling. (A) and (B) Representative images and statistical data show the expression of pPERK, PERK, <t>pIRE1α,</t> <t>IRE1α,</t> pNF-κB, NF-κB, pIP3R, IP3R, NCX, Kv1.5, and β-actin in HL-1 cells from control group (n = 3), TMAO treated (n = 3), SB treated (n = 3), and TMAO combined with SB treated group (n = 3) in three independent experiments. β-Actin was used as a loading control. * P < 0.05. ** P < 0.01. TMAO versus control or SB or TMAO combined with SB treatment.
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    Image Search Results


    SB supplement ameliorated TMAO-mediated activation of ER stress signaling and dysregulation of ionic signaling. (A) and (B) Representative images and statistical data show the expression of pPERK, PERK, pIRE1α, IRE1α, pNF-κB, NF-κB, pIP3R, IP3R, NCX, Kv1.5, and β-actin in HL-1 cells from control group (n = 3), TMAO treated (n = 3), SB treated (n = 3), and TMAO combined with SB treated group (n = 3) in three independent experiments. β-Actin was used as a loading control. * P < 0.05. ** P < 0.01. TMAO versus control or SB or TMAO combined with SB treatment.

    Journal: Journal of Advanced Research

    Article Title: Short-chain fatty acid butyrate against TMAO activating endoplasmic-reticulum stress and PERK/IRE1-axis with reducing atrial arrhythmia

    doi: 10.1016/j.jare.2024.08.009

    Figure Lengend Snippet: SB supplement ameliorated TMAO-mediated activation of ER stress signaling and dysregulation of ionic signaling. (A) and (B) Representative images and statistical data show the expression of pPERK, PERK, pIRE1α, IRE1α, pNF-κB, NF-κB, pIP3R, IP3R, NCX, Kv1.5, and β-actin in HL-1 cells from control group (n = 3), TMAO treated (n = 3), SB treated (n = 3), and TMAO combined with SB treated group (n = 3) in three independent experiments. β-Actin was used as a loading control. * P < 0.05. ** P < 0.01. TMAO versus control or SB or TMAO combined with SB treatment.

    Article Snippet: Then, the blots were incubated with primary antibodies against phospho-PERK (pPERK; #3179, Cell signaling), PERK (#3192, Cell signaling), phospho-IRE1α (pIRE1α; NB100-2323, Novus), IRE1α (#3294, Cell signaling Technology), phospho-NF-κB (pNF-κB; #3033, Cell Signaling Technology), NF-κB (#8242, Cell Signaling Technology), phospho-IP3R (pIP3R; #3760, Cell signaling Technology), IP3R (#8568, Cell signaling Technology), NCX (MA3-926, Thermo Fisher Scientific), Kv1.5 (APC-004, Alomone Lab), and β-actin (sc-47778, Santa Cruz).

    Techniques: Activation Assay, Expressing, Control

    SB supplement ameliorated atrial fibrosis and PERK/IRE1α/NF-κB axis of TMAO-treated mice. The expression of atrial fibrosis (blue area) was detected by Masson's staining and immunostaining of pPERK, pIRE1α, and pNF-κB in the atrium tissues derived from mice gavage with control (n = 5), TMAO (n = 5), and TMAO combined with SB (n = 5). The expressions of pPERK are indicated by hollow arrowheads. (bar, 25 μm). ** p < 0.01. TMAO treatment versus control group or TMAO combined with SB treated group. (For interpretation of the references to colour in this Fig. legend, the reader is referred to the web version of this article.)

    Journal: Journal of Advanced Research

    Article Title: Short-chain fatty acid butyrate against TMAO activating endoplasmic-reticulum stress and PERK/IRE1-axis with reducing atrial arrhythmia

    doi: 10.1016/j.jare.2024.08.009

    Figure Lengend Snippet: SB supplement ameliorated atrial fibrosis and PERK/IRE1α/NF-κB axis of TMAO-treated mice. The expression of atrial fibrosis (blue area) was detected by Masson's staining and immunostaining of pPERK, pIRE1α, and pNF-κB in the atrium tissues derived from mice gavage with control (n = 5), TMAO (n = 5), and TMAO combined with SB (n = 5). The expressions of pPERK are indicated by hollow arrowheads. (bar, 25 μm). ** p < 0.01. TMAO treatment versus control group or TMAO combined with SB treated group. (For interpretation of the references to colour in this Fig. legend, the reader is referred to the web version of this article.)

    Article Snippet: Then, the blots were incubated with primary antibodies against phospho-PERK (pPERK; #3179, Cell signaling), PERK (#3192, Cell signaling), phospho-IRE1α (pIRE1α; NB100-2323, Novus), IRE1α (#3294, Cell signaling Technology), phospho-NF-κB (pNF-κB; #3033, Cell Signaling Technology), NF-κB (#8242, Cell Signaling Technology), phospho-IP3R (pIP3R; #3760, Cell signaling Technology), IP3R (#8568, Cell signaling Technology), NCX (MA3-926, Thermo Fisher Scientific), Kv1.5 (APC-004, Alomone Lab), and β-actin (sc-47778, Santa Cruz).

    Techniques: Expressing, Staining, Immunostaining, Derivative Assay, Control